Sunday, May 31, 2009
The singer had an "emotional breakdown" following Saturday's final in which she was runner-up. But the talent show favorite was still eyeing a United States tour. The 48-year-old virgin, tipped to earn about $13 million, survived tears and a tantrum to finish second in Saturday's gripping final of television's "Britain's Got Talent." But the pressure finally showed late yesterday as the Scottish singer — dubbed SuBo by fans was rushed to the private clinic suffering from exhaustion. Show aides had contacted police to say she was acting strangely at her London hotel. Paramedics helped the "spaced-out" star through the lobby and into an ambulance just after 6 p.m. local time. A Met Police Inspector and a police doctor were called to assist. Show aides had contacted police to say she was acting strangely at her London hotel. Paramedics helped the "spaced-out" star through the lobby and into an ambulance just after 6 p.m. local time. A Met Police Inspector and a police doctor were called to assist. The ambulance, tailed by a police car, then took her to the Priory clinic in Southgate, North London. A source at the hotel said last night: "She'd been at the hotel for a few days, but since Saturday's final had been acting strangely, causing a bit of a stir. "The staff were concerned - something wasn't right. "When the paramedics and police arrived she agreed to go voluntarily. She didn't make a fuss. The paramedics calmly took her out through the main lobby and into the waiting ambulance. "It was all done very calmly. They didn't want to stress or upset her. She didn't look well - she looked lost, not all there." A source at the clinic said last night: "I was having a cigarette break when a whole load of ambulances arrived. "Everyone was saying, 'Who's that'? Then I saw her and it was Susan Boyle. I was gobsmacked."
Thursday, May 28, 2009
Wednesday, May 27, 2009
Tuesday, May 26, 2009
Sunday, May 24, 2009
INDIANAPOLIS — Three-fourths of the way through the 200-lap Indianapolis 500 on Sunday, pole-sitter Helio Castroneves had regained the lead. Castroneves led the first seven laps, then fell back as far as fifth. But he passed defending champion Scott Dixon after a caution period. Will Power, hired as a backup to Castroneves for Team Penske, was third, followed by Dan Wheldon and Townsend Bell. Ed Carpenter was sixth through 150 laps, followed by Danica Patrick, Paul Tracy, Ryan Briscoe and rookie Mike Conway. Tony Kanaan was running third when he crashed out of the race on lap 98. Jim Nabors had barely finished singing the traditional "Back Home Again (in Indiana)" when two drivers made a quick exit from the race. After the original start of the race was waved off because the field wasn't bunched, the cars of Mario Moraes and Marco Andretti made contact in the very first turn. Moraes' car was taken back to the garage area on a wrecker; Andretti's was receiving repairs in the pits. “The kid doesn’t get it; he never will,” Andretti said of the 20-year-old Moraes. “All the work, all the hopes, it’s over just like that. This place can just be so hard on you.” Moraes and Andretti started the race in the third row, having qualified seventh and eighth, respectively. But they never got up to speed on Sunday. “It’s so disappointing,” said Andretti. “The guy (Moraes) is clueless.” Said Moraes: “I was in front, I was holding my line, and he just hit me.” Andretti was hoping to win the race on the 40th anniversary of his grandfather Mario's lone victory in 1969. On lap 55, another of the young stars of IndyCar racing crashed out. Graham Rahal, son of 1986 winner Bobby Rahal, hit the wall hard for the second consecutive year. He started fourth and was running sixth, but was placed 28th on the grid after the crash. Last year, as a rookie, he started 13th but was the first car out with a crash on lap 36. The stands were full at Indianapolis beneath partly cloudy skies.
Friday, May 22, 2009
Tuesday, May 19, 2009
Monday, May 18, 2009
The United States Mint will mint and issue four different 2009 Lincoln pennies in recognition of the bicentennial birth of President Abraham Lincoln and the 100th anniversary of the Lincoln cent. While the obverse or heads side of the penny remains unchanged, the new reverse designs beautifully portray facets of Lincoln’s life, with the first cent officially launched into circulation on Feb. 12, at the Abraham Lincoln Birthplace National Historic Site, in Hodgenville, Kentucky.
2009 Lincoln Pennies
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When the first 2009 penny was issued on Lincoln’s birthday, Feb. 12, it marked the first redesign of the cent in 50 years. At the time the 2009 Lincoln cent designs were first revealed, United States Mint Director Ed Moy said:
“This is a momentous occasion in the history of our Nation’s coinage because these designs represent the first change in the Lincoln cent in half a century.
These coins are a tribute to one of our greatest Presidents whose legacy has had a lasting impact on our country. He believed all men were created equal, and his life was a model for accomplishing the American dream through honesty, integrity, loyalty, and a lifetime of education.”
Each penny will be released into general circulation at approximately three-months intervals throughout 2009. Although the US Mint has not officially declared exact release dates, the expected launch dates for the second, third and fourth penny are May 14, Aug. 13 and Nov. 12.
At the conclusion of the 2009 Lincoln Bicentennial Program — years 2010 and beyond — the one-cent coin will feature a reverse design that will be "emblematic of President Lincoln’s preservation of the United States of America as a single and united country."
2009 Lincoln Silver Dollars further honor America’s revered leader. The United States Mint has struck collector proof and uncirculated silver coins with a limited total mintage of 500,000.
2009 Lincoln Silver Dollars
The Lincoln coins were authorized through the Presidential $1 Coin Act of 2005, which was signed into law (Public Law 109-145) on Dec. 22, 2005 by President George Bush.
Sunday, May 17, 2009
Saturday, May 16, 2009
Friday, May 15, 2009
Thursday, May 14, 2009
Why do I need a Heel Stick test for my baby?
Although your living family may not have certain genetic disorders it is not out of the realm of possibility that a recessive gene may have become active in your child. The test can prevent permanent disability, mental retardation and in some cases death.
How are Heel Stick tests performed?
The Heel Stick test is done soon after you child is born, usually within the first 48 hours. Your baby’s heel is pricked and a few drops of blood are taken for analysis; the nurse collects nine or ten drops of blood and ships the sample to a lab to get tested. It is often wise to get your baby tested multiple times in order to produce the most accurate results.What disorders are tested for?
There are five categories of disorders that are screened:
Amino acid metabolism disorders
Babies with amino acid metabolism disorders, as the name suggests, have problems metabolizing certain amino acids. Babies born with amino acid metabolism disorders have missing or defective enzymes that metabolize proteins. The amino acids either are not broken down or the body cannot transport the amino acids into the cells. As a result, amino acids or nitrogen (a component of amino acids) build up in the body to toxic levels. Amino acid metabolism disorders include Phenylketonuria(PKU), Maple syrup urine disease (MSUD), Homocystinuria (HCY), Citrullinemia (CIT), Argininosuccinic academia (ASA), and Tyrosinemia type I (TYR I).
Organic Acid Metabolism Disorders
Babies with organic acid metabolism disorders cannot process certain branched amino acids. Babies born with this disorder do not produce the enzymes or have malfunctioning enzymes that do not break down these amino acids properly. As a result, organic acids build up to toxic level in the body. Some organic acid metabolism disorders that your baby might be screened for are Isovaleric academia (IVA), Glutaric acidemia type I (GA I), and Hydroxymethylglutaric aciduria or HMG-CoA lyase (HMG).
Fatty Acid Oxidation Disorders
Fatty acid oxidation refers to the way your body breaks down fat. Fat is broken down in several steps and requires a number of enzymes. If one of the enzymes in this process is not working properly the body cannot turn fat into energy for the body. The body’s main source of energy is glucose, but if there is no glucose available, your body turns to fat for energy. Babies with fatty acid oxidation disorders have malfunctioning enzymes so their bodies cannot turn fat into energy. Some examples of fatty acid oxidation disorders that your baby might be screened for are Long Chain 3-Hydroxyacyl-CoA Dehydrogenase Deficiency (LCHAD), Medium Chain Acyl-CoA Dehydrogenase Deficiency (MCAD), Short Chain Acyl-CoA Dehydrogenase Deficiency (SCAD), Trifunctional Protein Deficiency (TFP Deficiency), or Very Long Chain Acyl-CoA Dehydrogenase Deficiency (VLCAD).
Hemoglobinopathies and sickle cell disease
Hemoglobinopathies and sickle cell disease are disorders caused by abnormal hemoglogin, a protein in red blood cells that is responsible for transporting oxygen to the body. Babies with these hemoglobinopathies may have problems with red blood cell production resulting in anemia (shortage of red blood cells) or produce abnormally shaped red blood cells that cause problems in blood circulation and damage easily. These disorders include sickle cell anemia, Thalassemia, and Hb S/C disease.
Other disorders your baby might be screened for include Congenital hypothyroidism (a hormone deficiency that causes problems with growth and brain development), Biotinidase deficiency, Congenital adrenal hyperplasia (an endocrine disorder that affects hormone production from the adrenal gland), or Cystic Fibrosis (a life threatening disease that causes mucous secretions in the lungs to become thick causing severe respiratory problems).
If your baby tests positive during the Heel Stick screening you will need to bring your child in for additional testing. The tests are very sensitive so false positives are a common occurrence. You may need to consult a genetic specialist or pediatrician to entirely diagnose your child.